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Table 1 Neurophysiological data

From: Early impairment of intracranial conduction time predicts mortality in deeply sedated critically ill patients: a prospective observational pilot study

Evoked potentials data Healthy control subjects (n = 20) Deeply sedated critically ill patients (n = 86)
Mean latency ± SD (in ms) Mean latency + 2.5 SD (in ms) Mean latency ± SD (in ms) Delayed latency n (%)
SSEP components
 N9 PL 9.8 ± 0.6 11.4 11.0 ± 1.6 33 (38)
 N13 PL 13.2 ± 0.8 15.2 14.9 ± 2.1 36 (42)
 P14 PL 14.6 ± 0.9 16.8 16.2 ± 2.0 32 (37)
 N20 PLa 18.8 ± 1.0 21.4 21.6 ± 2.6 41 (48)
 N9-N13 IPL 3.4 ± 0.4 4.4 3.9 ± 0.9 14 (16)
 P14-N20 IPLa
(ICCT)
4.1 ± 0.5 5.3 5.3 ± 1.5 39 (45)
BAEP components
 Wave I PL 1.5 ± 0.2 2.0 1.69 (0.31) 13 (15)
 Wave III PL 3.6 ± 0.2 4.1 3.96 (0.35) 34 (40)
 Wave V PL 5.6 ± 0.3 6.3 6.16 (0.61) 34 (40)
 I–III IPL 2.1 ± 0.2 2.6 2.28 (0.29) 17 (20)
 III–V IPL (IPCT) 2.0 ± 0.2 2.5 2.20 (0.45) 15 (17)
 I–V IPL 4.1 ± 0.3 4.8 4.51 (0.59) 17 (20)
  1. PL peak latency, IPL inter-peak latency, ICCT intracranial conduction time, IPCT intra- pontine conduction time. PL and IPL of SSEP or BAEP’s components were scored as “delayed” when they were greater than the “mean + 2.5 SD” of the ones of a healthy control group
  2. aN20 was abolished in three patients. N20 PL and P14-N20 IPL were consequently considered as delayed for these 3 patients with abolished N20. Median nerve somatosensory evoked potentials (SSEP) and brainstem auditory evoked potentials (BAEP) components latencies (PL) and inter-peak latencies (IPL) obtained from a group of 20 healthy control subjects (11 women, 9 men, mean age 51 ± 17 years) in our laboratory; and from 86 deeply sedated critically ill patients. Each variable is represented by the mean values of left and right hand sides